Vitamin C treatment reduces elevated C-reactive protein

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Vitamin C treatment reduces elevated C-reactive protein.

Plasma C-reactive protein (CRP) is an inflammatory biomarker that predicts cardiovascular disease. Lowering elevated CRP with statins has reduced the incidence of cardiovascular disease. We investigated whether vitamin C or E could reduce CRP. Healthy nonsmokers (N=396) were randomized to three groups, 1000 mg/day vitamin C, 800 IU/day vitamin E, or placebo, for 2 months. Median baseline CRP wa...

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Effects of Vitamin C Supplementation on C-Reactive Protein, Lactate and Blood Pressure Following Resistance Exercise in Overweight Men

Background and Objectives: Strenuous exercises have been shown to induce inflammation. However, consumption of products rich in antioxidants such as vitamin C may enhance this effect. The purpose of this study was to assess effects of vitamin C supplementation on C-reactive protein, lactate and blood pressure following resistance exercise in overweight men. Materials and Methods: In a randomiz...

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Rosuvastatin in patients with elevated C-reactive protein.

To the Editor: Ridker et al. (Nov. 20 issue) report the results of Justification for the Use of Statins in Prevention: an Intervention Trial Evaluating Rosuvastatin (JUPITER),1 which showed that rosuvastatin significantly reduced cardiovascular events in patients with elevated levels of C-reactive protein. However, broad application of their results in primary prevention is premature, since the...

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Rosuvastatin in Patients with Elevated C-Reactive Protein

To the Editor: Ridker et al. (Nov. 20 issue) report the results of Justification for the Use of Statins in Prevention: an Intervention Trial Evaluating Rosuvastatin (JUPITER),1 which showed that rosuvastatin significantly reduced cardiovascular events in patients with elevated levels of C-reactive protein. However, broad application of their results in primary prevention is premature, since the...

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ژورنال

عنوان ژورنال: Free Radical Biology and Medicine

سال: 2009

ISSN: 0891-5849

DOI: 10.1016/j.freeradbiomed.2008.09.030